Melatonin inhibits glucocorticoid receptor nuclear translocation in mouse thymocytes

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The antiapoptotic effect of melatonin (MEL) has been described in several systems. In particular, MEL inhibits glucocorticoid-mediated apoptosis. Our group previously demonstrated that in the thymus, MEL inhibits the release of Cytochrome C from mitochondria and the dexamethasone-dependent increase...

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Detalles Bibliográficos
Autores principales: Presman, D.M., Hoijman, E., Ceballos, N.R., Galigniana, M.D., Pecci, A.
Formato: Artículo publishedVersion
Publicado: 2006
Materias:
cell nucleus receptor
cytochrome c
dexamethasone
glucocorticoid receptor
heat shock protein 90
indole derivative
melatonin
messenger RNA
methoxyindole
orphan nuclear receptor ROR alpha
protein Bax
retinoid
unclassified drug
animal cell
animal experiment
article
breast epithelium
controlled study
drug inhibition
drug mechanism
gene expression
gene translocation
immunoprecipitation
ligand binding
male
mouse
nonhuman
priority journal
thymocyte
transcription initiation
Active Transport, Cell Nucleus
Animals
Apoptosis
Cell Nucleus
Dexamethasone
Male
Melatonin
Mice
Protein Transport
Receptors, Glucocorticoid
T-Lymphocytes
Thymus Gland
Acceso en línea:http://hdl.handle.net/20.500.12110/paper_00137227_v147_n11_p5452_Presman
https://repositoriouba.sisbi.uba.ar/gsdl/cgi-bin/library.cgi?a=d&c=artiaex&d=paper_00137227_v147_n11_p5452_Presman_oai
Aporte de:
Repositorio Digital de la Universidad de Buenos Aires (UBA) de Universidad de Buenos Aires

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