Tunicamycin inhibits the initiation of DNA synthesis stimulated by prostaglandin F2α in Swiss mouse 3T3 cells

Tunicamycin, an inhibitor of the asparagine-linked protein N-glycosylation, blocks the initiation of DNA synthesis in Swiss 3T3 cells stimulated by prostaglandin F2α alone or with insulin. This effect is exerted only when tunicamycin is added from 0 to 8 h after stimulation and it decreases the rate...

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Detalles Bibliográficos
Publicado:	1991
Materias:	DNA synthesis Lag phase Prostaglandin F2α Swiss 3T3 cell Tunicamycin insulin prostaglandin f2 alpha tunicamycin animal cell article cell growth dna synthesis mouse nonhuman priority journal 3T3 Cells Animal Cell Division Dinoprost DNA Glucosamine Glycoproteins In Vitro Insulin Mannose Mice Support, Non-U.S. Gov't Time Factors Animalia
Acceso en línea:	https://bibliotecadigital.exactas.uba.ar/collection/paper/document/paper_00145793_v290_n1-2_p239_Estevez http://hdl.handle.net/20.500.12110/paper_00145793_v290_n1-2_p239_Estevez
Aporte de:	Biblioteca Digital - Facultad de Ciencias Exactas y Naturales (UBA) de Universidad de Buenos Aires

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