Lowering arterial pressure delays the oxidative stress generation in a renal experimental model of hypertension
Oxidative stress produced through reactive oxygen species (ROS) enhancement is considered to play a key role in the development and maintenance of hypertension. In the vasculature, the most important source of ROS is the reduced nicotinamide adenine dinucleotide phosphate (NAD(P)H) oxidase enzyme. T...
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Formato: | JOUR |
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Acceso en línea: | http://hdl.handle.net/20.500.12110/paper_01602446_v54_n4_p348_Polizio |
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