Gallo, G., Valko, A., Aramburu, S., Etchegaray, E., Völker, C., Parodi, A., & D’Alessio, C. (2018). Abrogation of glucosidase I–mediated glycoprotein deglucosylation results in a sick phenotype in fission yeasts: Model for the human MOGS-CDG disorder. American Society for Biochemistry and Molecular Biology Inc. https://doi.org/10.1074/jbc.RA118.004844
Cita Chicago Style (17a ed.)Gallo, G.L, A. Valko, S.I Aramburu, E. Etchegaray, C. Völker, A.J Parodi, y C. D’Alessio. Abrogation of Glucosidase I–mediated Glycoprotein Deglucosylation Results in a Sick Phenotype in Fission Yeasts: Model for the Human MOGS-CDG Disorder. American Society for Biochemistry and Molecular Biology Inc, 2018. https://dx.doi.org/10.1074/jbc.RA118.004844.
Cita MLA (8a ed.)Gallo, G.L, et al. Abrogation of Glucosidase I–mediated Glycoprotein Deglucosylation Results in a Sick Phenotype in Fission Yeasts: Model for the Human MOGS-CDG Disorder. American Society for Biochemistry and Molecular Biology Inc, 2018.