p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine

Celiac disease (CD) is a chronic enteropathy elicited by a Th1 response to gluten peptides in the small intestine of genetically susceptible individuals. However, it remains unclear what drives the induction of inflammatory responses of this kind against harmless antigens in food. In a recent work,...

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Autores principales: Gómez Castro, María Florencia, Miculán, Emanuel, Herrera, María Georgina, Ruera, Carolina, Pérez, Federico, Prieto, Eduardo Daniel, Barrera, Exequiel, Pantano, Sergio, Carasi, Paula, Chirdo, Fernando Gabriel
Formato: Articulo
Lenguaje:Inglés
Publicado: 2019
Materias:
Acceso en línea:http://sedici.unlp.edu.ar/handle/10915/107861
http://europepmc.org/backend/ptpmcrender.fcgi?accid=PMC6363691&blobtype=pdf
Aporte de:
id I19-R120-10915-107861
record_format dspace
institution Universidad Nacional de La Plata
institution_str I-19
repository_str R-120
collection SEDICI (UNLP)
language Inglés
topic Biología
Enteropathy
Celiac disease
Inflammasome
Caspase-1
p31-43
Gliadin peptides
Innate immunity
Small intestine damage
spellingShingle Biología
Enteropathy
Celiac disease
Inflammasome
Caspase-1
p31-43
Gliadin peptides
Innate immunity
Small intestine damage
Gómez Castro, María Florencia
Miculán, Emanuel
Herrera, María Georgina
Ruera, Carolina
Pérez, Federico
Prieto, Eduardo Daniel
Barrera, Exequiel
Pantano, Sergio
Carasi, Paula
Chirdo, Fernando Gabriel
p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine
topic_facet Biología
Enteropathy
Celiac disease
Inflammasome
Caspase-1
p31-43
Gliadin peptides
Innate immunity
Small intestine damage
description Celiac disease (CD) is a chronic enteropathy elicited by a Th1 response to gluten peptides in the small intestine of genetically susceptible individuals. However, it remains unclear what drives the induction of inflammatory responses of this kind against harmless antigens in food. In a recent work, we have shown that the p31-43 peptide (p31-43) from α-gliadin can induce an innate immune response in the intestine and that this may initiate pathological adaptive immunity. The receptors and mechanisms responsible for the induction of innate immunity by p31-43 are unknown and here we present evidence that this may reflect conformational changes in the peptide that allow it to activate the NLRP3 inflammasome. Administration of p31-43, but not scrambled or inverted peptides, to normal mice induced enteropathy in the proximal small intestine, associated with increased production of type I interferon and mature IL-1β. P31-43 showed a sequence-specific spontaneous ability to form structured oligomers and aggregates in vitro and induced activation of the ASC speck complex. In parallel, the enteropathy induced by p31-43 in vivo did not occur in the absence of NLRP3 or caspase 1 and was inhibited by administration of the caspase 1 inhibitor Ac-YVAD-cmk. Collectively, these findings show that p31-43 gliadin has an intrinsic propensity to form oligomers which trigger the NLRP3 inflammasome and that this pathway is required for intestinal inflammation and pathology when p31-43 is administered orally to mice. This innate activation of the inflammasome may have important implications in the initial stages of CD pathogenesis.
format Articulo
Articulo
author Gómez Castro, María Florencia
Miculán, Emanuel
Herrera, María Georgina
Ruera, Carolina
Pérez, Federico
Prieto, Eduardo Daniel
Barrera, Exequiel
Pantano, Sergio
Carasi, Paula
Chirdo, Fernando Gabriel
author_facet Gómez Castro, María Florencia
Miculán, Emanuel
Herrera, María Georgina
Ruera, Carolina
Pérez, Federico
Prieto, Eduardo Daniel
Barrera, Exequiel
Pantano, Sergio
Carasi, Paula
Chirdo, Fernando Gabriel
author_sort Gómez Castro, María Florencia
title p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine
title_short p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine
title_full p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine
title_fullStr p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine
title_full_unstemmed p31-43 Gliadin Peptide Forms Oligomers and Induces NLRP3 Inflammasome/Caspase 1- Dependent Mucosal Damage in Small Intestine
title_sort p31-43 gliadin peptide forms oligomers and induces nlrp3 inflammasome/caspase 1- dependent mucosal damage in small intestine
publishDate 2019
url http://sedici.unlp.edu.ar/handle/10915/107861
http://europepmc.org/backend/ptpmcrender.fcgi?accid=PMC6363691&blobtype=pdf
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