Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice

Abstract: Patients with obstructive sleep apnea (OSA) exhibit a high prevalence of pulmonary hypertension and right ventricular (RV) hypertrophy. However, the exact molecule responsible for the pathogenesis remains unknown. Given the resistance to RV dilation observed in transient receptor potent...

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Autores principales: Park, Do Yang, Heo, Woon, Kang, Miran, Ahn, Taeyoung, Kim, DoHyeon, Choi, Ayeon, Birnbaumer, Lutz, Cho, Hyung Ju, Kim, Joo Young
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Publicado: MDPI 2023
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Acceso en línea:https://repositorio.uca.edu.ar/handle/123456789/17375
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spelling I33-R139-123456789-173752023-11-23T18:11:58Z Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice Park, Do Yang Heo, Woon Kang, Miran Ahn, Taeyoung Kim, DoHyeon Choi, Ayeon Birnbaumer, Lutz Cho, Hyung Ju Kim, Joo Young APNEA OBSTRUCTIVA DEL SUEÑO HIPOXIA CRONICA INTERMITENTE POTENCIAL RECEPTOR TRANSITORIO CANAL CATIONICO 3 VENTRICULO DERECHO HIPERTROFIA DEL VENTRICULO DERECHO DILATACION DEL VENTRICULO DERECHO ENDOTELINA Abstract: Patients with obstructive sleep apnea (OSA) exhibit a high prevalence of pulmonary hypertension and right ventricular (RV) hypertrophy. However, the exact molecule responsible for the pathogenesis remains unknown. Given the resistance to RV dilation observed in transient receptor potential canonical 3(Trpc3)−/− mice during a pulmonary hypertension model induced by phenylephrine (PE), we hypothesized that TRPC3 also plays a role in chronic intermittent hypoxia (CIH) conditions, which lead to RV dilation and dysfunction. To test this, we established an OSA mouse model using 8- to 12-week-old 129/SvEv wild-type and Trpc3−/− mice in a customized breeding chamber that simulated sleep and oxygen cycles. Functional parameters of the RV were evaluated through analysis of cardiac cine magnetic resonance images, while histopathological examinations were conducted on cardiomyocytes and pulmonary vessels. Following exposure to 4 weeks of CIH, Trpc3−/− mice exhibited significant RV dysfunction, characterized by decreased ejection fraction, increased end-diastole RV wall thickness, and elevated expression of pathological cardiac markers. In addition, reactive oxygen species (ROS) signaling and the endothelin system were markedly increased solely in the hearts of CIH-exposed Trpc3−/− mice. Notably, no significant differences in pulmonary vessel thickness or the endothelin system were observed in the lungs of wild-type (WT) and Trpc3−/− mice subjected to 4 weeks of CIH. In conclusion, our findings suggest that TRPC3 serves as a regulator of RV resistance in response to pressure from the pulmonary vasculature, as evidenced by the high susceptibility to RV dilation in Trpc3−/− mice without notable changes in pulmonary vasculature under CIH conditions. 2023-10-31T11:37:14Z 2023-10-31T11:37:14Z 2023 Artículo Park, D. Y. et al. Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice [en línea]. International Journal of Molecular Sciences. 2023, 24(14), 11284. doi: 10.3390/ijms241411284. Disponible en: https://repositorio.uca.edu.ar/handle/123456789/17375 1422-0067 https://repositorio.uca.edu.ar/handle/123456789/17375 10.3390/ijms241411284 37511045 eng Acceso abierto http://creativecommons.org/licenses/by-nc-sa/4.0/ application/pdf MDPI International Journal of Molecular Sciences. Vol. 24, No.14, 11284, 2023
institution Universidad Católica Argentina
institution_str I-33
repository_str R-139
collection Repositorio Institucional de la Universidad Católica Argentina (UCA)
language Inglés
topic APNEA OBSTRUCTIVA DEL SUEÑO
HIPOXIA CRONICA INTERMITENTE
POTENCIAL RECEPTOR TRANSITORIO CANAL CATIONICO 3
VENTRICULO DERECHO
HIPERTROFIA DEL VENTRICULO DERECHO
DILATACION DEL VENTRICULO DERECHO
ENDOTELINA
spellingShingle APNEA OBSTRUCTIVA DEL SUEÑO
HIPOXIA CRONICA INTERMITENTE
POTENCIAL RECEPTOR TRANSITORIO CANAL CATIONICO 3
VENTRICULO DERECHO
HIPERTROFIA DEL VENTRICULO DERECHO
DILATACION DEL VENTRICULO DERECHO
ENDOTELINA
Park, Do Yang
Heo, Woon
Kang, Miran
Ahn, Taeyoung
Kim, DoHyeon
Choi, Ayeon
Birnbaumer, Lutz
Cho, Hyung Ju
Kim, Joo Young
Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice
topic_facet APNEA OBSTRUCTIVA DEL SUEÑO
HIPOXIA CRONICA INTERMITENTE
POTENCIAL RECEPTOR TRANSITORIO CANAL CATIONICO 3
VENTRICULO DERECHO
HIPERTROFIA DEL VENTRICULO DERECHO
DILATACION DEL VENTRICULO DERECHO
ENDOTELINA
description Abstract: Patients with obstructive sleep apnea (OSA) exhibit a high prevalence of pulmonary hypertension and right ventricular (RV) hypertrophy. However, the exact molecule responsible for the pathogenesis remains unknown. Given the resistance to RV dilation observed in transient receptor potential canonical 3(Trpc3)−/− mice during a pulmonary hypertension model induced by phenylephrine (PE), we hypothesized that TRPC3 also plays a role in chronic intermittent hypoxia (CIH) conditions, which lead to RV dilation and dysfunction. To test this, we established an OSA mouse model using 8- to 12-week-old 129/SvEv wild-type and Trpc3−/− mice in a customized breeding chamber that simulated sleep and oxygen cycles. Functional parameters of the RV were evaluated through analysis of cardiac cine magnetic resonance images, while histopathological examinations were conducted on cardiomyocytes and pulmonary vessels. Following exposure to 4 weeks of CIH, Trpc3−/− mice exhibited significant RV dysfunction, characterized by decreased ejection fraction, increased end-diastole RV wall thickness, and elevated expression of pathological cardiac markers. In addition, reactive oxygen species (ROS) signaling and the endothelin system were markedly increased solely in the hearts of CIH-exposed Trpc3−/− mice. Notably, no significant differences in pulmonary vessel thickness or the endothelin system were observed in the lungs of wild-type (WT) and Trpc3−/− mice subjected to 4 weeks of CIH. In conclusion, our findings suggest that TRPC3 serves as a regulator of RV resistance in response to pressure from the pulmonary vasculature, as evidenced by the high susceptibility to RV dilation in Trpc3−/− mice without notable changes in pulmonary vasculature under CIH conditions.
format Artículo
author Park, Do Yang
Heo, Woon
Kang, Miran
Ahn, Taeyoung
Kim, DoHyeon
Choi, Ayeon
Birnbaumer, Lutz
Cho, Hyung Ju
Kim, Joo Young
author_facet Park, Do Yang
Heo, Woon
Kang, Miran
Ahn, Taeyoung
Kim, DoHyeon
Choi, Ayeon
Birnbaumer, Lutz
Cho, Hyung Ju
Kim, Joo Young
author_sort Park, Do Yang
title Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice
title_short Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice
title_full Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice
title_fullStr Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice
title_full_unstemmed Role of TRPC3 in right ventricular dilatation under chronic intermittent hypoxia in 129/SvEv mice
title_sort role of trpc3 in right ventricular dilatation under chronic intermittent hypoxia in 129/svev mice
publisher MDPI
publishDate 2023
url https://repositorio.uca.edu.ar/handle/123456789/17375
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